<p class="jove_content">PM2.5 exposure induces delayed testicular injury in rat models through sustained inflammatory responses, enhanced germ cell apoptosis, downregulation of Connexin-43/Occludin, and signaling consistent with activation of the JNK pathway, highlighting its long-term threat to male reproductive health.</p>

delayed-progressive-post-exposure-testicular-injury-rats-due-to-fine

Delayed and Progressive Post Exposure Testicular Injury in Rats Due to Fine Particulate Matter (PM2.5)

Fine particulate matter (PM2.5) is increasingly implicated in male reproductive impairment. However, because most research remains acute-focused, delayed, ...

Institution - Fujian health college

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